Mold exposure may interfere with thiamine in more than one way. This clip explores how certain mycotoxins may directly affect B1, while mold-related neuroinflammation can also disrupt thiamine chemistry downstream. It also looks at reports of improved brain function from lipid-soluble thiamine derivatives, even in people still dealing with mold exposure.
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What If Parkinson’s Is Also An Energy Metabolism Problem?
Parkinson’s is often framed around dopamine, but this clip looks deeper at what may be happening inside the cell. Oxidative stress, inflammation, chronic infection, and environmental toxins can interfere with key thiamine-dependent enzymes involved in energy production, including pyruvate dehydrogenase and alpha-ketoglutarate dehydrogenase. That raises a bigger question about whether impaired B1-dependent metabolism is part of the disease process itself.
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