DS Haven In Light Of Things
High-dose thiamine is not always about correcting a simple dietary deficiency. This clip explains how very large doses may help reactivate thiamine-dependent enzymes that have become blocked or impaired, allowing cells to produce energy more effectively. The discussion also points to traumatic brain injury research showing how restoring B1-dependent energy pathways may reduce secondary cellular damage.
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What If Parkinson’s Is Also An Energy Metabolism Problem?
Parkinson’s is often framed around dopamine, but this clip looks deeper at what may be happening inside the cell. Oxidative stress, inflammation, chronic infection, and environmental toxins can interfere with key thiamine-dependent enzymes involved in energy production, including pyruvate dehydrogenase and alpha-ketoglutarate dehydrogenase. That raises a bigger question about whether impaired B1-dependent metabolism is part of the disease process itself.